Update 'datacite.yml'

datacite.yml

@@ -74,8 +74,8 @@ description: |
   protects against cognitive impairment induced by experimental perinatal asphyxia. To investigate the 
   long-term neuropathological effects of hypoxic-ischemic injury to the developing brain and the role 
   of C3aR signaling therein, we subjected wildtype mice, C3aR deficient mice, and mice expressing 
-  biologically active C3a in the CNS to mild hypoxic-ischemic brain injury on postnatal day 9. 
-  We found that such injury triggers neurodegeneration and pronounced reactive gliosis in the ipsilesional 
+  biologically active C3a in the CNS to mild hypoxic-ischemic brain injury on postnatal day 9. We found 
+  that such injury triggers neurodegeneration and pronounced reactive gliosis in the ipsilesional 
   hippocampus both of which persist long into adulthood. Transgenic expression of C3a in reactive astrocytes
   reduced hippocampal neurodegeneration and reactive gliosis. In contrast, neurodegeneration and microglial 
   cell density increased in mice lacking C3aR. Intranasal administration of C3a for 3 days starting